Gain Therapeutics Unveils GT-02287 Parkinson’s Therapy Data at Neuroscience 2025

Reuters

10/30

Gain <a href="https://laohu8.com/S/LENZ">Therapeutics</a> Unveils GT-02287 Parkinson's Therapy Data at Neuroscience 2025

Gain Therapeutics, Inc., a clinical-stage biotechnology company, announced it will present new research on its small molecule allosteric modulator, GT-02287, at the upcoming Society for Neuroscience annual meeting, Neuroscience 2025. The poster presentation, scheduled for November 19, 2025, will focus on the therapeutic potential of GT-02287, which targets the lysosomal enzyme glucocerebrosidase (GCase) and demonstrates activity at both lysosomes and mitochondria. The results of this research will be presented at the conference and have not yet been disclosed.

Disclaimer: This news brief was created by Public Technologies (PUBT) using generative artificial intelligence. While PUBT strives to provide accurate and timely information, this AI-generated content is for informational purposes only and should not be interpreted as financial, investment, or legal advice. Gain Therapeutics Inc. published the original content used to generate this news brief via GlobeNewswire (Ref. ID: GNW9565041-en) on October 30, 2025, and is solely responsible for the information contained therein.

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The poster presentation, scheduled for November 19, 2025, will focus on the therapeutic potential of GT-02287, which targets the lysosomal enzyme glucocerebrosidase (GCase) and demonstrates activity at both lysosomes and mitochondria. The results of this research will be presented at the conference and have not yet been disclosed.\n      </p>\n</div><div xmlns:xsd=\"http://www.w3.org/2001/XMLSchema\" xmlns:xsi=\"http://www.w3.org/2001/XMLSchema-instance\">\n<em>Disclaimer: <span>This news brief was created by Public Technologies (PUBT) using generative artificial intelligence. While PUBT strives to provide accurate and timely information, this AI-generated content is for informational purposes only and should not be interpreted as financial, investment, or legal advice. Gain Therapeutics Inc. published the original content used to generate this news brief via GlobeNewswire (Ref. ID: GNW9565041-en) on October 30, 2025, and is solely responsible for the information contained therein.</span></em>\n</div></body></html>\n\n</article>\n</div>\n</body>\n</html>\n","isBrief":false,"type":0,"news_type":1,"symbol":"BK4112","symbol_name":"金融交易所和数据","start_time":0,"source_url":"https://api.refinitiv.com/data/news/v1/stories/urn:newsml:reuters.com:20251030:nNDL4h4dhB:1","article_id":"2579117897","we_media_id":"1032215980","thumbnails":[],"rights":null,"url":"https://stock-news.laohu8.com/highlight/detail?id=2579117897","pubTimestamp":1761822034,"columns":[],"sourceInfo":null,"weMediaInfo":{"media_name":"Reuters","introduction":"Reuters.com brings you the latest news from around the world, covering breaking news in markets, business, politics, entertainment and technology","home_visible":1,"id":"1032215980","head_image":"https://community-static.tradeup.com/news/4567337cbdf294b657b1fa87c5488b48"},"summary":"Gain Therapeutics, Inc., a clinical-stage biotechnology company, announced it will present new research on its small molecule allosteric modulator, GT-02287, at the upcoming Society for Neuroscience annual meeting, Neuroscience 2025. 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The poster presentation, scheduled for November 19, 2025, will focus on the therapeutic potential of GT-02287, which targets the lysosomal enzyme glucocerebrosidase (GCase) and demonstrates activity at both lysosomes and mitochondria. The results of this research will be presented at the conference and have not yet been disclosed.\n      \n\nDisclaimer: This news brief was created by Public Technologies (PUBT) using generative artificial intelligence. While PUBT strives to provide accurate and timely information, this AI-generated content is for informational purposes only and should not be interpreted as financial, investment, or legal advice. Gain Therapeutics Inc. published the original content used to generate this news brief via GlobeNewswire (Ref. 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